Search results for "Nude mouse"

showing 8 items of 8 documents

Preclinical xenograft models of human sarcoma show nonrandom loss of aberrations

2011

BACKGROUND: Human tumors transplanted into immunodeficient mice (xenografts) are good preclinical models, and it is important to identify possible systematic changes during establishment and passaging in mice. METHODS: High-resolution microarray-based comparative genomic hybridization (array CGH) was used to investigate how well a series of sarcoma xenografts, including 9 patient/xenograft pairs and 8 early versus late xenograft passage pairs, represented the patient tumor from which they originated. RESULTS: In all analyses, the xenografts were more similar to their tumor of origin than other xenografts of the same type. Most changes in aberration patterns were toward a more normal genome …

Cancer ResearchPathologymedicine.medical_specialtyMicroarraybiologyCancerPDGFRAbiology.organism_classificationmedicine.diseaseTransplantationNude mouseOncologyTumor progressionmedicineSarcomaComparative genomic hybridizationCancer
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Hybrid Chelator-Based PSMA Radiopharmaceuticals: Translational Approach

2021

(1) Background: Prostate-specific membrane antigen (PSMA) has been extensively studied in the last decade. It became a promising biological target in the diagnosis and therapy of PSMA-expressing cancer diseases. Although there are several radiolabeled PSMA inhibitors available, the search for new compounds with improved pharmacokinetic properties and simplified synthesis is still ongoing. In this study, we developed PSMA ligands with two different hybrid chelators and a modified linker. Both compounds have displayed a promising pharmacokinetic profile. (2) Methods: DATA5m.SA.KuE and AAZTA5.SA.KuE were synthesized. DATA5m.SA.KuE was labeled with gallium-68 and radiochemical yields of various…

Diagnostic ImagingGlutamate Carboxypeptidase IIBiodistributionmedia_common.quotation_subjectPharmaceutical ScienceOrganic chemistryChemistry Techniques Syntheticurologic and male genital diseasesArticleAnalytical ChemistryTranslational Research BiomedicalMicechemistry.chemical_compoundhybrid chelatorNude mouseQD241-441In vivoNeoplasmsDrug DiscoveryLNCaPAnimalsHumansChelationradionuclide diagnosis and therapyPhysical and Theoretical ChemistryInternalizationChelating Agentsmedia_commonMolecular StructurebiologyChemistryRadiochemistrybiology.organism_classificationDisease Models AnimalKineticsChemistry (miscellaneous)Isotope LabelingAntigens SurfaceHeterograftsMolecular MedicineRadiopharmaceuticalsAmmonium acetateEx vivoprostate specific membrane antigen PSMAProtein BindingMolecules
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Inhibition of DNA methylation sensitizes glioblastoma for tumor necrosis factor-related apoptosis-inducing ligand-mediated destruction.

2005

AbstractLife expectancy of patients affected by glioblastoma multiforme is extremely low. The therapeutic use of tumor necrosis factor–related apoptosis-inducing ligand (TRAIL) has been proposed to treat this disease based on its ability to kill glioma cell lines in vitro and in vivo. Here, we show that, differently from glioma cell lines, glioblastoma multiforme tumors were resistant to TRAIL stimulation because they expressed low levels of caspase-8 and high levels of the death receptor inhibitor PED/PEA-15. Inhibition of methyltransferases by decitabine resulted in considerable up-regulation of TRAIL receptor-1 and caspase-8, down-regulation of PED/PEA-15, inhibition of cell growth, and …

MaleCancer ResearchMethyltransferaseNudeDrug ResistanceApoptosisReceptors Tumor Necrosis FactorTNF-Related Apoptosis-Inducing LigandCASPASE-8 EXPRESSIONMiceNude mouseSIGNALING COMPLEXReceptorsAntineoplastic Combined Chemotherapy ProtocolsTumor Cells CulturedDNA Modification MethylasesIN-VIVOHeterologousCaspase 8CulturedMembrane GlycoproteinsbiologyIntracellular Signaling Peptides and ProteinsMiddle AgedTumor CellsGene Expression Regulation NeoplasticMALIGNANT GLIOMA-CELLSOncologyCaspasesDNA methylationAzacitidineTumor necrosis factor alphaFemalemedicine.drugSignal TransductionAdultBRAIN-TUMORSTransplantation HeterologousCHEMOTHERAPEUTIC-AGENTSDecitabineMice NudeDecitabineDRUG-INDUCED APOPTOSISDEATH RECEPTOR5-AZA-2'-DEOXYCYTIDINEIn vivoSettore MED/04 - PATOLOGIA GENERALEmedicineAnimalsHumansneoplasmsAgedTransplantationNeoplasticCell growthTumor Necrosis Factor-alphaHistocompatibility Antigens Class IDNA Methylationbiology.organism_classificationPhosphoproteinsReceptors TNF-Related Apoptosis-Inducing LigandGene Expression RegulationApoptosisDrug Resistance NeoplasmImmunologyCancer researchNeoplasmAdult; Aged; Animals; Antineoplastic Combined Chemotherapy Protocols; Apoptosis; Apoptosis Regulatory Proteins; Azacitidine; Caspase 8; Caspases; DNA Modification Methylases; Drug Resistance Neoplasm; Female; Glioblastoma; Histocompatibility Antigens Class I; Humans; Intracellular Signaling Peptides and Proteins; Male; Membrane Glycoproteins; Mice; Mice Nude; Middle Aged; Phosphoproteins; Receptors TNF-Related Apoptosis-Inducing Ligand; Receptors Tumor Necrosis Factor; Signal Transduction; TNF-Related Apoptosis-Inducing Ligand; Transplantation Heterologous; Tumor Cells Cultured; Tumor Necrosis Factor-alpha; DNA Methylation; Gene Expression Regulation Neoplastic; Cancer Research; OncologyTumor Necrosis FactorTRAIL-INDUCED APOPTOSISApoptosis Regulatory ProteinsGlioblastomaCancer research
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Cryoablation of Human Colorectal Cancerin Vivoin a Nude Mouse Xenograft Model

1998

Abstract Objective: To establish the minimum required temperature in cryoablation of human colorectal cancer cell lines grown as subcutaneous tumors in mice. Methods: Male nu/nu nude mice were inoculated by a sc injection of 1 × 10 6 LoVo ( n = 30) or C170 ( n = 32) cells. After 2 weeks the tumors were frozen using a 3-mm cryotherapy probe (LCS 3000, Cryotech, UK) to temperatures ranging from −8 to −84°C. Results: (LoVo) Of 21 mice evaluable for analysis no tumors recurred in 3 mice which had their tumors frozen to less than −60°C as measured at the presumed tumor/host boundary, whereas all but one tumor recurred in 18 mice which had their tumors frozen to >−60°C. (C170) Of 18 mice evaluabl…

MalePathologymedicine.medical_specialtyRatónColorectal cancermedicine.medical_treatmentTransplantation HeterologousMice NudeRectumCryotherapyGeneral Biochemistry Genetics and Molecular BiologyCryosurgeryMiceNude mousemedicineAnimalsHumansbiologybusiness.industryCryoablationNeoplasms ExperimentalGeneral Medicinebiology.organism_classificationmedicine.diseaseCold Temperaturemedicine.anatomical_structureCryotherapyCell cultureColorectal NeoplasmsGeneral Agricultural and Biological SciencesbusinessNeoplasm TransplantationCryobiology
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Virus and Experimental Diabetes

1984

Special subtypes of viruses belonging to different virus families are known to be able to induce insulin-deficient diabetes or pathological glucose tolerance in experimental animals (Table 1). The development of the diabetogenic effect of viruses is dependent on the species (Table 1) and, within one species, dependent on age and sex (Friedman et al., 1972), particularly on the genetic factors of the animals determining susceptibility or resistance to diabetes (Craighead and Higgins, 1974; Yoon and Notkins, 1976).

Nude mouseDiabetes mellitusImmunologymedicineBiologyAge and sexmedicine.diseasebiology.organism_classificationPathologicalVirusVirus classificationExperimental diabetes
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New cell lines of gastric and pancreatic cancer: distinct morphology, growth characteristics, expression of epithelial and immunoregulatory antigens.

1995

Two new cell lines from stomach cancers and one from a pancreatic carcinoma are presented. MZ-GC-1 was established from a hepatic metastasis of a well differentiated gastric adenocarcinoma. MZ-GC-2 was derived from ascites induced by a poorly differentiated gastric adenocarcinoma. MZ-PC-1 originated from the pleural effusion of a moderately well differentiated pancreatic ductal adenocarcinoma. MZ-GC-1 cells were adherent and partially polarized, connected tightly via desmosomes. In contrast MZ-GC-2 cells consisted of slightly adherent or floating subpopulations and displayed no desmosomes. MZ-PC-1 cells were adherent and showed polarized growth, connected by apical junctional complexes. Cel…

Pathologymedicine.medical_specialtyCell divisionCellular differentiationCellBiologyAdenocarcinomaEpitheliumPathology and Forensic MedicineCytokeratinNude mouseStomach NeoplasmsPancreatic cancermedicineBiomarkers TumorHumansNeoplasm MetastasisMolecular BiologyCell Line TransformedLiver NeoplasmsCell DifferentiationCell BiologyGeneral Medicinebiology.organism_classificationmedicine.diseasePancreatic NeoplasmsMicroscopy Electronmedicine.anatomical_structureCell cultureAntigens SurfaceCancer researchMicroscopy Electron ScanningPancreasCell DivisionVirchows Archiv : an international journal of pathology
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Biliary adenocarcinoma

1985

Three human cell lines from adenocarcinomas of the extrahepatic biliary tract were established in permanent tissue culture. Mz-ChA-1 and Mz-ChA-2 were cultured from mechanically dissociated gallbladder adenocarcinoma metastases and SK-ChA-1 was grown from malignant ascites of a patient with primary adenocarcinoma of the extrahepatic biliary tree. Cell doubling times in tissue culture are 3-4 days for Mz-ChA-1 and approximately 2 days for Mz-ChA-2 and SK-ChA-1. All three tumour cell lines were successfully transplanted to nude mice, inducing progressive tumour growth. Histologically, nude mouse tumours resembled the original adenocarcinomas. In vitro formation of gland-like structures were r…

Pathologymedicine.medical_specialtyHepatologyeducationCellHepatobiliary diseaseBiologymedicine.diseasebiology.organism_classificationTransplantationTissue cultureNude mousemedicine.anatomical_structureCell cultureparasitic diseasesmedicineAdenocarcinomaNeoplasmJournal of Hepatology
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Head and Neck Cancers

2005

Pyriform SinusNude mousebiologybusiness.industryFalse Vocal CordTonsillar fossaMedicineAnatomyHead and neckbusinessbiology.organism_classification
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